This month I would like to present an intriguing case of dementia. Neural therapy was not used in treatment, but an interference field was detected in the small intestine by autonomic response testing. And this proved to be the key to both diagnosis and successful treatment.
There was more to this case than simply dementia. The patient actually presented with night time seizures, but it was the decline in memory that was the most worrying, because the patient's father had suffered from early‐onset Alzheimer's disease. Here is the story:
An otherwise healthy 64‐year old man presented with grand mal seizures, always at 4 am and beginning two years previously. At first they occurred every three months, but then became more frequent and severe. During this period both the patient and his wife noticed a gradual decline in memory.
Many investigations resulted in no diagnosis and the patient was prescribed Levetiracetam shortly before I first saw him. Because of the lack of diagnosis, the patient and his wife sought out alternative therapies in another country and the patient underwent four sessions of intravenous EDTA chelation ‐ which seemed to help his memory.
On his first visit, a medical and structural examination was unremarkable. A search for interference fields using autonomic response testing (or ART) in the cranial vault, brainstem, teeth, and viscera detected nothing. Because of his positive clinical response to EDTA chelation, an intravenous DMPS provocation test was performed with only minimal amounts of mercury and lead appearing on urinalysis. .
However, routine blood testing showed one unexpected finding: a mild eosinophilia of .700 cells/μL. Eosinophilia is in my practice a "red flag" for intestinal parasites and/or Lyme disease. On the next visit, a more careful check for interference fields was undertaken and one was found in the small intestine. Homeopathics for various infectious organisms were tested by ART and a response was obtained to Herpes Type 1 virus, with reversal of the response in the presence of colloidal silver (Mesosilver) and EPA (Eicosapentanoic acid). The patient was prescribed both Mesosilver and EPA.
Three months later, his memory was improving and at 10 months his memory and cognition was "back to normal". However 1 1/2 years later he returned with his memory again deteriorating. The interference field in the small intestine had also returned with herpes virus the causative agent. This time there was no autonomic response to colloidal silver, but fortunately there was one to the herb Samento (cat's claw). Two months of Samento and his brain function was again restored to normal.
Although this patient had a dramatic improvement in his dementia with treatment by colloidal silver (and later Samento), a number of questions arise: Why the small intestine with neurological symptoms? Was the herpes doing its damage in the gut or in the brain? How does colloidal silver work anyway?
While researching these questions I stumbled across some ideas that might explain not only how this patient got better, but also provide fresh insight into our understanding of how neural therapy actually works.
The work I am referring to is that of W John Martin, a controversial pathologist and immunologist who has developed a theory of stealth viruses ("stealth" because they do not trigger an immune response). In addition, he postulates that there exists an "alternative cellular energy" pathway that is damaged by stealth viruses, but can be repaired by various "activating" substances including colloidal silver and procaine! Classical neural therapy theory recognizes that procaine is a sodium channel blocker and that it treats interference fields by repolarizing partly de-polarized cell membranes. But perhaps there is more to procaine than that.
If in fact procaine repairs damaged "alternative cellular energy" pathways, we may have to rethink the nature of interference fields. They may be far more complex than we have thought for these many years. Dr Papathanasiou (newsletter Vol.8, no.10) has already proposed that interference fields are capable of signalling the immune and endocrine systems. And autonomic response testing allows "questioning" of the interference field for information about what is causing it, why it is persisting and even remedies for correcting it.
The case presented above leaves many unanswered questions. Can a "stealth" virus explain the patient's symptoms? If so, why did the interference field present in the gut? We know there is a strong brain‐gut connection, but this patient had no gastrointestinal complaints. What was the cause of the eosinophilia? Could there have been a parasite co‐infection? Does this patient have tick‐borne illness (Lyme disease)?
I apologise to readers for posing more questions than providing answers, but I would be interested to hear if anyone knows about stealth viruses and especially if they have found an interface with neural therapy.
Robert F. Kidd, MD, CM