One of the best indications that neural therapy may be the solution to our patient's pain problem is failure of manual treatments to help a situation that is clearly mechanical in nature. This is especially evident when chronic pain develops after a traumatic event and skilled manual treatment gives no lasting relief.
My patient, (a 42 year old female) developed neckache not long after a front-end motor vehicle collision two years before her appointment. The pain had responded poorly to chiropractic manipulation and massage, and in fact headaches had begun in the five months preceding her visit. The neck pain centered in the right upper posterior cervical region and the headache was felt in the right occiput and right forehead. Activity provoked pain in the right interscapular area. Clicking was experienced in the right tempero-mandibular area, but this symptom had been present even before the accident.
The patient otherwise had an uncomplicated medical history. Her wisdom teeth had been extracted in her teens and she had undergone a hysterectomy at age 26 for mennorhagia. There was no other history of trauma. However, she had noticed some decline in her energy over the previous year and attributed this to mould exposure in her workplace, as she felt better on the weekends.
On examination there was a restriction of rotation of the head to the right and a palpable area of localized muscle spasm about 3 cm in diameter just to the right of the C2 and C3 vertebrae. The right temporal bone was externally rotated. All three Ayurvedic pulses (See vol.6, no.2 of my newsletters) on the right side were very weak, indicating an important interference field somewhere on the right side of the body. In addition, a "therapy localization sign" was present on the right side (see page 51 of my book). Deep tendon reflexes throughout the body were symmetric and quite brisk.
The above signs were highly suggestive of an interference field in the head or neck region and a careful search was made using autonomic response testing (See chapter 4 of my book.) The wisdom teeth scars, tonsils and teeth were checked, as well as the cervical sympathetic ganglia (because of the whiplash injury). Nothing was found until the cranial autonomic ganglia were tested. An interference field was detected in the right submandibular ganglion and it matched the therapy localization sign - a strong indicator that this was the interference field. This was puzzling as submandibular ganglia interference fields are usually associated with dental problems.
Treatment with a Tenscam device, (an alternative would have been to inject 3 ml. procaine ½% - see page 183 of my book) resulted in abolition of the interference field, but no change in the blocked regulation. This indicated that there was still "unfinished business". A further search indicated an interference field in the right sphenopalatine ganglion.This was also a puzzle because sphenopalatine ganglia usually become "activated" with dental or facial sinus problems. However, it crossed my mind that the patient's sphenopalatine ganglion interference field might have something to do with the patient's fatigue and reaction to mould in her workplace. (The brisk tendon reflexes might also have been caused by the mould's neurotoxic effects.) A specimen of mould was then introduced into her field, and the autonomic response (indicator muscle weakening) reversed, supporting the idea that mould was at least part of the reason for this up‐regulation of her nervous system. The sphenopalatine ganglion was then treated with the Tenscam device and open regulation resulted. The patient was discharged with advice to reduce her exposure to mould.
Three weeks later the patient reported that her headaches were "less severe". Re‐examination revealed again an interference field in the right submandibular ganglion, this time associated with an interference field in tooth 4.7 (second right lower molar). Using dental homeopathics, it became apparent there was an occult infection in or near the tooth. This was treated using the ultraviolet frequency of the Tenscam device; the interference field disappeared, and regulation was again achieved.
On the next visit one week later, the headaches and neckache had disappeared entirely, but tooth 4.7 needed one final treatment to achieve open regulation. Two weeks later the patient presented with no neckache or headache, no interference fields, and open regulation.
This case was interesting for a number of reasons: (1) the non‐mechanical factors preventing resolution of a seemingly mechanical injury; (2) the (at first) completely hidden dental interference field: (3) and the contribution of an environmental factor (the mould). (Mould is a neurotoxin in some people and can have similar effects on the nervous system as have toxic metals, gluten, organic solvents and other toxins.)
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Dear Dr. Kidd,
I was inspired to share a 'skin patient' with you based on your most recent Newsletter.
I have a 40 year old female patient who had been experiencing ongoing skin issues for over a year ‐ the diagnosis was folliculitis. I traced back the onset to around the time of a dog bite on the woman's thigh. We treated the scar with neural therapy. I didn't see the patient for over a year, never knowing the outcome of the treatment. I just saw her last month and she stated that after the treatment her skin issues resolved and she has been touting the excellence of my care ever since. Teaches me that following up is so important to know how effective or ineffective our treatments are.
Thank‐you for sharing your passion for neural therapy.
Dr. Jennifer Moss,
Robert F. Kidd, MD, CM